Endometrial cancer is one of the almost popular cancers in American women. In endometrial cancer, cancer cells produce in the lining of the womb. Why these cancer cells produce is not completely known. However, scientists think that estrogen levels beat a character in the growth of endometrial cancer. Endometrial cancer may develop in breast cancer patients who have been treated with tamoxifen. Women taking estrogen alone have an increased risk of developing endometrial cancer. Taking estrogen in combination with progesterone does not increase a woman’s risk of this cancer. Endometrial cancer is sometimes called uterine cancer, but there are other cells in the uterus that can become cancerous - such as muscle or myometrial cells. These form much less common cancers called sarcomas and account for less than 5 percent of uterine cancers.
Endometrial cancer is frequently detected at an earlier phase because it often produces vaginal hemorrhage between menstrual periods or after menopause. Some of the same danger factors for bosom cancer and ovarian cancer too increase the danger of endometrial cancer. Endometrial cancer usually begins in the endometrium, the lining of the uterus - a hollow, pear-shaped pelvic organ where fetal development occurs. If discovered early, this slow-growing cancer is likely to be confined to the uterus. White women are more likely to develop endometrial cancer, but black women are much more likely to die of the disease. Diabetes is a risk factor for endometrial cancer mainly because obesity and type 2 diabetes often go hand in hand. Estrogen stimulates growth of the endometrium. Replacing estrogen alone after menopause may increase the risk of endometrial cancer.
Endometrial cancer frequently develops over a period of years. Most cases of endometrial cancer produce in postmenopausal women, whose periods have stopped.
Rarely does endometrial cancer hit a sophisticated phase before any signs and symptoms are existing. Conversely, some women who develop endometrial cancer appear to have no risk factors for the disease. Because endometrial cancer is usually diagnosed in the early stages, there is a better probable outcome associated with it than with other types of gynecological cancers such as cervical or ovarian cancer. Use of oral contraceptives can reduce endometrial cancer risk even as long as 10 years after you stop taking them. The risk is lowest in women who take oral contraceptives for many years. A woman with advanced endometrial cancer may have other symptoms, such as losing weight without trying.
Surgery is the almost popular main handling for endometrial cancer. Surgical handling should comprise of, at least, cytologic sample of the peritoneal fluid, abdominal exploration, palpation and biopsy of suspicious lymph nodes, abdominal hysterectomy, and removal of both ovaries. If the cancer has spread to other parts of your body, synthetic progestin, a form of the hormone progesterone, may stop it from growing.
Chemotherapy is the use of drugs to kill cancer cells.
In some cases, your doctor may recommend chemotherapy for endometrial cancer. These drugs enter your bloodstream and then travel through your body, killing cancer cells outside the uterus. Abdominal hysterectomy is recommended over vaginal hysterectomy because it affords the opportunity to examine and obtain washings of the abdominal cavity to detect any further evidence of cancer.